top | item 30028376

(no title)

podgaj | 4 years ago

It’s about controlling the out of control inflammation caused by the body fighting the infection.

discuss

order

netizen-936824|4 years ago

No it isn't. From the study:

>The mechanism appears to be mediated in part by activation of the IRE1α RNase and interferon pathways.

>Thus, the RNase activity of IRE1α induced by CBD can potentially account both for the degradation of viral RNA and the induction of interferons by the RNA fragments.

podgaj|4 years ago

Yes, it is. They are not explaining HOW IRE1a is triggered by CDB, and it is because CDB triggers the CB1/2 receptor which activates these pathways.

https://journals.plos.org/plosone/article?id=10.1371/journal...

"Overall, our study investigates for the first time the effects of the endocannabinoid AEA on primary human T-lymphocytes, demonstrating that it is a powerful modulator of immune cell functions. In particular, not only we clarify that CB2R mediates the immunosuppressive activity of AEA, but we are the first to describe such an immunosuppressive effect on the newly identified Th-17 cells. These findings might be of crucial importance for the rational design of new endocannabinoid-based immunotherapeutic approaches."

The only way CBD can trigger these pathways is by effecting a cell receptor The only cell receptors for CBD are CB1r and CB2R. There is literally no other way this can happen.

If y9ou can explain how CBD increases IRE1α activity any other way I am all ears,